A new research has suggested that the belief that majority of long term speech and language problems are due to negligent parenting skill is far from accurate.

The survey, conducted by Communication Trust, polled more than 6,000 people, which included over 3,000 parents, and found that over 50 percent of the parents believe that the problems are caused due to poor communication with their children.

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It looked at how the rise in deaths after the loss related to age and sex and race, and found that in women, there was a 27 percent increase in all circulatory deaths.

In men, there was a 15 percent increase in such deaths associated with the loss.

“The Super Bowl may elicit an emotional response that is similar in US females and males, or perhaps a male’s reaction to the Super Bowl loss adversely affected the emotional state of a female partner,” the study said.

Women also fared better than men after the Super Bowl win four years later.

“For women, but not men, there was a reduction in all-cause death and circulatory deaths associated with the Super Bowl win,” the study said.

After the loss, more cardiac deaths occurred overall across sex barriers. And among people over 65, there was a 22 percent increase in circulatory deaths, though no statistically significant differences were found among various races.

“Physicians and patients should be aware that stressful games might elicit an emotional response that could trigger a cardiac event,” said lead study author Robert Kloner.

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The researchers have discovered a key clue as to why this happens, by understanding how the ‘gates,’ which effectively ‘open’ and ‘close’ the channel, operate.

“Just like a set of metal wires that carry electricity to light up our streets, our body has a series of channels that carry tiny charged particles called ions, into and out of cells, to trigger a heartbeat,” said Jamie Vandenberg, Head of the Cardiac Electrophysiology Laboratory at the Victor Chang Institute.

“Depending on the position of these gates, many common drugs bind, or attach themselves to these channels, blocking the ions from passing through. This causes what we call Long QT syndrome, where the length of the heart beat is longer than usual, which greatly increases the risk of arrhythmia,” said Vandenberg.

The team of researchers, led by Vandenberg, studied the hERG potassium channel, an ion channel that determines how long each heart beat lasts and the channel which is most susceptible to being ‘blocked’ by drugs.The hERG channel is a particularly ‘sticky’ channel, in that most drugs will bind to it when the outer gate is closed. What we’ve done is to discover how these outer gates operate, in the hope that we can then design drugs more effectively to minimise the unwanted side effects.

Article source: http://feedproxy.google.com/~r/allhealthnews/~3/s89MaN7k0AU/Gates-on-Sudden-Cardiac-Death-Unlocked-by-Scientists-80162-1.htm

Transcranial magnetic stimulation (TMS) is based on the fact that the cortex, the rind of the brain located directly underneath the skull bone, can be stimulated by means of a magnetic field.

One single magnetic pulse serves to test the activability of nerve cells in an area of the cortex, in order to assess changes in diseases or after consumption of medications or also following a prior artificial stimulation of the brain.

Medical experts from Bochum under the leadership of Prof. Dr. Klaus Funke (Department of Neurophysiology) have now shown for the first time that an artificial cortex stimulation specifically changes the activity of certain inhibitory nerve cells as a function of the stimulus protocol used.

Funke’s group was also able to show that rats also learned more quickly if they were treated with the activating stimulus protocol (iTBS) before each training.

Repetitive TMS is already being used in clinical trials with limited success for therapy of functional disorders of the brain, above all in severe depressions.

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“The fetal inflammatory response appears to contribute to the onset of preterm labor, fetal injury and complications, underlying lifetime health challenges facing these children,” said the researchers from Harvard Medical School, Brigham and Women’s Hospital and Children’s Hospital of Boston.

“Our data suggest that placental colonization by specific groups of organisms can increase or decrease the risk of a systemic inflammatory condition,” the authors said.

The systemic fetal inflammatory response to intrauterine exposures, especially intrauterine infections, is regarded as an important contributor to the onset and often life-long consequences of preterm labor, fetal injury and early organ damage.

Approximately half of all placentas delivered before the second trimester and 41percent of those delivered by Caesarean section harbor microorganisms detectable by culture techniques.

In order to better understand what role, these microorganisms could play in the extremely preterm inflammatory response the researchers analyzed protein biomarkers in dry blood spots obtained from 527 newborns delivered by Caesarean section and cultured and identified the bacteria from their respective placentas.

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Immunosuppressive drugs can form a part of treatment of autoimmune diseases like rheumatoid arthritis, lupus or HIV, which   arise from inappropriate immune responses. In such instances, the body’s immune system attacks the body, instead of attacking foreign cells. It is to inhibit such a response, the immunosuppressives are pressed into service.  Such drugs are also used in organ transplant.

When an organ, such as a liver, a heart or a kidney, is transplanted from one person (the donor) into another (the recipient), the immune system of the recipient triggers the same response against the new organ it would have to any foreign material, setting off a chain of events that can damage the transplanted organ. The immunosuppressant drugs greatly decrease the risks of rejection, protecting the new organ and preserving its function.

Now a study at the Indiana University School of Medicine determined that the antibiotic compound tautomycetin targets an enzyme called SHP2, which plays an important role in cell activities such as proliferation and differentiation. Interestingly, SHP2 mutations are also known to cause several types of leukemia and solid tumors. The findings were reported in the Jan. 28, 2011, issue of the journal Chemistry and Biology.

The potential for developing anti-cancer agents grew out of an attempt to determine how the compound, tautomycetin, exerts its immune suppression activities, said Zhong-Yin Zhang, Ph.D., Robert A. Harris Professor and chairman of the Department of Biochemistry and Molecular Biology.

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